News & Publications

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NEWS

Posters

AAIC 2017 - BBB

16-20 JULY 2017, LONDON
Effects of circulating and neuronal amyloid peptide oligomers on the BBB integrity : consequences on the neurodegeneration process.

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AD/PD 2017 - PD

29 MARCH - 02 APRIL 2017, VIENNA (Austria)
MPP+ induced dopaminergic neuron death through necroptosis and autophagy.

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AD/PD 2017 - ALS

29 MARCH - 02 APRIL 2017, VIENNA (Austria)
Amyloid peptide and cytoplasmic TDP-43 accumulation in pathogenesis of ALS : an in vitro study.

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MNM 2016 - AD/BBB

29-30 SEPTEMBER 2016, LONDON
A novel triple cell co-culture model for mimicking the BBB into normal or pathological situations.

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AAIC 2016 - AD

22 - 28 JULY 2016, TORONTO
Mode of action study of NSP01-E, a potential treatment for Alzheimer’s disease, in protection against amyloid β peptide (Aβ) injury.

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MYOLOGY 2016 - ALS

14-18 MARCH 2016, LYON (France)
Pathological role of amyloid peptide in neuromuscular disease, glutamate and glucose involvement.

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JSFM 2015 - ALS

23-25 NOVEMBER 2015, LYON (France)
Deleterious effects of amyloid beta peptide in the neuromuscular junction : consequences in ALS disease.

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PHYT’AROM 2015 - SNC-1

5-7 JUNE 2015, GRASSE (France)
Neuroprotective effect of SNC-1 a natural plant extract. Promising treatment for Alzheimer’s and Parkinson’s disease ?

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AD/PD 2015 - PD

MARCH 2015, NICE (France)
α-synuclein : cause or consequence of cytopathological lesions observed in in vitro models of Parkinson’s disease.

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AD/PD 2015 - AD

MARCH 2015, NICE (France)
Neurodegeneration induced by Aβ oligomeric fractions : role of NMDA receptors and mGluR5.

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ARUK CONF. 2015 - AD

MARCH 2015, LONDON
NSP01-E01 and NSP01-C01 : new preparations able to protect from beta-amyloid peptide (Aβ) injuries. Potential treatments for Alzheimer’s disease.

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SFN 2014 - PD

NOVEMBER 2014, WASHINGTON
Study of mitochondrial death pathways in different cellular models of Parkinson’s disease : effect of 17β-Estradiol.

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SFN 2014 - AD

NOVEMBER 2014, WASHINGTON
Beta-estradiol protects cortical neurons from beta-amyloid oligomers toxicity through mediations of the Ras/raf and PI3K pathways.

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ICNMD 2014 - ALS

JUNE 2014, NICE (France)
Role of β-Amyloid (Aβ) Oligomeric fractions in ALS disease.

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Publications